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380 Vorinostat also reduces human immunodeficiency virus (HIV) replication through autophagy activation in infected macrophages without cytotoxicity, 350 supporting the findings of clinical trials for HIV latency reversal (such as NCT01365065, NCT01319383, NCT01933594, and NCT01680094)

In parallel, mTOR inhibitors such as rapamycin enhance lysosomal biogenesis and autophagic flux through activation of TFEB, which has been shown in animal studies to reduce granulosa cell apoptosis and promote follicular development [354]

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And while KPV suppresses inflammation with precision, it does not build new blood vessels, stimulate collagen production, or structurally repair tissue that has already been damaged