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doi: 10.1016/j.toxlet.2019.04.014 86 KeunenKVan ElburgRMVan BelFBendersMJ
CP 200 mg/kg caused hepatotoxicity due to oxidative stress, inflammation, apoptosis, and fibrosis as manifested by a reduction in catalase, reduced glutathione (GSH), superoxide dismutase (SOD), and an increase in thiobarbituric acid reactive substance (TBARS), nitrite, tumor necrosis factor-alpha (TNF-), interleukin-6 (IL-6), transforming growth factor-beta 1 (TGF-1), interleukin-1 (IL-1), alkaline phosphatase (ALP), alanine aminotransferase (ALT), aspartate aminotransferase (AST), and gamma-glutamyl transferase (GGT) levels
CEO has also been shown to mimic superoxide dismutase activity, as evidenced by its ability to inhibit pyrogallol autoxidation [81, 82]
Therefore, decreased mPFC taurine levels might limit the defensive capabilities of mPFC neurons and NAC could potentially compensate for this loss by enhancing GSH synthesis and rebalancing the local redox system
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