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All these alterations make IGF-1 Long R3 significantly more potent (2-3x) than IGF-1 in studies, because it has a lower affinity to be rendered inactive by IGF binding proteins

GABAergic deficits and glutamate excitotoxicity are two essential signaling pathways that could be addressed to treat autism, thus medications targeting these pathways are critical for treating behavioral symptoms

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There is also significant person-to-person variability two AD patients might have very different microbiome alterations yet end up with similar pathology, indicating multiple microbial paths to the same disease