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In addition, FGF21 treatment significantly reduces ACSL4 and iron deposition, increases GPX4 expression, and reduces ferroptosis in nerve cells by activating the FGFR1/-Klotho pathway

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An explanation for this association between excess cytoplasmic retention and ROS production has been put forward, based on the correlation between the latter and the cellular content of glucose-6-phosphoate dehydrogenase in human sperm suspensions [28]