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Conclusions Mechanistic and preliminary clinical evidence have consistently pointed toward beneficial effects of GLP-1 analogues and GLP-1R agonists on cardiovascular diseases in T2D patients

GLP-1 medications are peptides, and peptides are fragile biological molecules that start degrading the moment they leave controlled storage conditions

However, the answer may lie in the location and metabolic activity of the excessive adipose tissue in these patients

Studies have shown that semaglutide upregulates the expression of ITPR1 and ADCY5 in CRC by activating the GLP-1 receptor, which, in turn, inhibits the motility of CRC cells [88]