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Consistently, the motor nerve conduction study confirmed the absence of demyelinated processes in the tail caudal nerves after spinal cord injury (the CMAP showed normal biphasic potentials, similar amplitudes, and similar conduction velocities in all of the rats) (Table 4)

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This highlights the necessity of long-term antioxidant therapy to mitigate its effects (De Ornellas Strapazzon et al

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