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Mutants (A30P and A53T) were more likely translocated to the IMS than the wild-type, and more -Syn would be translocated to IMS upon oxidative stress or cI inhibition (Vicario et al., 2019)

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doi: 10.1111/j.1463-1326.2011.01376.x 55 FunchDGydesenHTorneKMajor-PedersenAChanKA

Though each compound acts through distinct biochemical pathways, their overlapping molecular targets reveal an integrative network capable of restoring insulin signaling, modulating steroidogenesis, and re-establishing ovulatory rhythm, reflecting a multidimensional redox-endocrine homeostasis that current pharmacotherapies only partially address