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Due to the extensive variability in the type, quality, and consistency of the articles available, a meta-analysis was not able to be conducted as this study was not able to adjust for the level of heterogenicity

Specific tissue types, including the brain and kidney, do not need insulin for glucose intake because the constitutive expression of the GLUT1 and GLUT3 transporters exists in these tissues [1]

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A second mechanism suggests that biased agonism, such as activity on the MC4R-G q pathway, may mitigate pressor effects while maintaining anorectic activity (66, 153)