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Indeed, vitamin E prevents de novo membrane oxidative damage, folate maintains levels of the endogenous antioxidant GSH and ALC prevents A-beta-induced mitochondrial damage and ATP depletion providing superior protection to that derived from each agent alone [151]

The disruption in mitochondrial activity, such as mitochondrial transfer from stromal cells to tumor cells, is crucial to tumor cell survival 34 , and the malate-aspartate shuttle constitutes a significant source of ROS 35

Similarly, NS oil decreased Bax/Bcl2 levels and down-regulated the expressions of caspase-3 (pro and cleaved forms) proteins in the brain and liver tissues of mice after d-galactose-induced aging (Shahroudi et al., 2017)