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Subsequently, mice with cardiomyocyte-specific (CM-specific) deletion of Glp1r also showed cardioprotective effects of liraglutide in an in vivo model of MI (22), suggesting that the cardioprotective effects of this agent are derived from actions independent of CMs and/or the GLP-1R

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Results The disruption of D2R-DAT stimulates locomotor activity As aforementioned, we developed an interfering peptide (TAT-DAT NT ) to disrupt the interaction between DAT and D2R, which can increase locomotor activity in mice [25]

Lyophilised format: Freeze-dried powder is the only appropriate format for research-grade storage and transit