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If residual schizophrenia reflects neural damage due to oxidative stress, reduced levels of glutamate and glutathione should be most marked in patients with residual schizophrenia

Gene expression signatures of C1-27 and GSTO1 knockdown To identify pathways regulated by GSTO1 and to understand the mechanism of its antiproliferative effects, we used the nascent RNA Bru-seq method 27 to profile gene expression changes that occur in response to GSTO1 knockdown and inhibition