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Tumor neutrophils ferroptosis: a targetable immunosuppressive mechanism for cancer immunotherapy

Accumulating evidence highlights that mitochondrial reactive oxygen species (ROS) imbalance is not merely a byproduct of tissue damage but acts as a central pathological driver, initiating and sustaining skeletal muscle wasting

13 Although evidence suggests that GLP-1 RAs may have neuroprotective properties and delay disease progression, this was based on a few small phase II studies