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For example, if two drugs share metabolic pathways, one might raise the levels of the other

Though she was prescribed medication, she didn't make lifestyle changes a move she feels might have "very slowly made my condition worse." The neuroscientist was also diagnosed with Sjgren's syndrome, dysautonomia, connective tissue disease, and mast cell activation syndrome (MCAS) in later years, and suffered from extreme symptoms of the conditions, including "crippling depression" and "full-body rashes." Bialik shared that separate three doctors suggested she go on a GLP-1 drug "not because of the 20 postmenopausal pounds but because the drugs have shown promise in reducing the systemic inflammation that drives autoimmune conditions." "Maybe this could be the magic cure," she recalled of her mindset at the time

the two conditions share common pathogenic mechanisms and influence each other Excessive glutamate release and activation of glutamate receptors promotes glioma growth, cell death and epileptic activity GABAergic signalling is antiproliferative, whereas chloride accumulation is required for mitosis and migration of tumour cells, and is responsible for epileptogenic depolarizing GABAergic activity in neurons The molecular target of rapamycin (mTOR) signalling pathway and epigenetic abnormalities are also involved in epileptogenesis and tumour growth As a result of the shared pathogenic mechanisms, antiepileptic drugs can have antitumour effects, and antitumour therapy can control seizures Single drug therapies targeting the shared mechanisms are now being assessed for combined seizure and tumour control, and have the advantage of lower risks of adverse effects and drug interactions Abstract Epilepsy often develops in patients with glioma, and the two conditions share common pathogenic mechanisms

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