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Endothelium-neutrophil interactions in ANCA-associated diseases

Overexpression of Rptor (a core component of mTORC1) in HMC-3 microglia blunted butyrates capacity to suppress ISR signaling and to restrain LD formation, establishing mTORC1 as the obligatory node in butyrate-mediated neuroprotection

Conclusions Based on our results, it can be concluded that CM might be used as a drug and functional food in inhibiting the oxidation and hyperlipidemia in an SD model of alcohol-induced hepatic disease possibly because of cordycepin and polyphenolic compound with potential anti-oxidative and anti-hyperlipidemic activities

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