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Some MS lesion sites are associated with impaired expression of mitochondrial ETC subunits and increased oxidative damage/stress (Maldonado et al., 2022) while other MS lesions exhibit an enhanced density of mitochondria and upregulated mitochondrial complex IV activity (Witte et al., 2009)
These barriers are physically represented by the so-called tight junctions between brain endothelial cells and epithelial cells, attributed to the special proteins such as occludin, claudins, and the associated proteins zona occludens (ZO-1, ZO-2, and ZO-3), which are highly expressed in brain endothelium (Chen et al., 2009
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