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These findings demonstrate that nitrosative stress, unlike ROS-mediated oxidation, directly targets redox-active cysteines of PDI, thereby interrupting the ER redox cycle

Stern completed her obstetrics residency in Albert Einstein College of Medicine in New York

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Mice given extra GLP-1 (via gene therapy or a GLP-1 agonist drug) developed more oxidative, fatigue-resistant muscle fibers (more Type I fibers), built up greater muscle glycogen stores, and increased mitochondrial biogenesis changes that enhanced their endurance performance (1)