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The NRF2-dependent effect was identified through the antioxidative response element in target gene loci, while NRF2-independent effects act through DNA 5hmC modifications via the regulation of the metabolite L2HG

(4) mGPDH in the regulation of ferroptosis G3P metabolism supports ferroptosis defense in disease contexts In the first study identifying mGPDH as a ferroptosis regulator, metabolomic analyses revealed that treatment of cancer cells with GPX4 inhibitors led to a marked depletion of intracellular glycerol3phosphate (G3P) 99

Redness or slight discomfort at the injection site, which usually fades quickly

p = 0.23 or the MT-II treated MIA mice