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Our findings demonstrated that GPX1 protected against peroxynitrite-induced SGNs damage by inhibiting oxidative stress and apoptosis, at least partially, through inhibiting the activation of nuclear factor-kappa B (NF-B) pathway
Given the multifactorial and heterogeneous pathophysiology of tinnitus, which involves maladaptive neuroplasticity, inflammation, oxidative stress, and often comorbid psychological distress, a single-target pharmacological intervention is unlikely to be universally effective
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doi: 10.1016/0163-7258(91)90076-X
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