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Abnormal expression and dysfunction of KARs are associated with various neurological diseases and disorders, including schizophrenia, mood disorders, epilepsy, and Huntingtons disease 8,9,10,11,12,13,14,15,16,17

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Although the underlying mechanisms are not fully understood, proposed contributors include reductions in N-terminal proB-type natriuretic peptide levels [95], improvements in cardiac remodeling [96], and anti-inflammatory effects [97]