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Serious problems can happen, and the long-term effects are unknown

Mounting evidence indicates that both disease biology and drug responsiveness in MASH and fibrosis are sexually dimorphic: pre-menopausal women exhibit slower fibrosis progression but may respond differently to metabolic therapies [55, 60, 61]

NOX4 isoform upregulates after viral infection in lung epithelial cells and is responsible for ROS generation, which activates protein kinases that favor viral ribonucleoprotein nuclear export and promote viral replication [31]

Effector T cells that secrete GM-CSF [8], Th17 cells that produce glutamate [9], CXCR4 + T cells with GM-CSF [10], regulatory T cells (Tregs) [11], CD8 + T lymphocytes [12], and regulatory B cells (B regs) that secrete IL-10 and IL-35 are only a few newly identified immune cells in MS [13, 14]