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Likewise, SARS-Cov-2 infection and acute COVID-19 are accompanied by an inflammatory response [29], increased MPO activity [45], indicants of oxidative damage [46,47,48] and increased NO production including increased levels of nitrotyrosine [49], and lowered antioxidant defenses (ANTIOX) as indicated by reduced TAC levels [45, 49], Gpx [50], and zinc [51]

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Nonetheless, PUFAs (such as arachidonic acid and epinephrine) are vulnerable to the Fenton reaction, which produces excessive peroxides that impair the phospholipid bilayer structure, thereby compromising cell membrane function [29,30,31]

After intravenous administration, the agents enter the systemic circulation and rapidly undergo covalent binding to plasma biomacromolecules within a short period, with human serum albumin (HSA) as the primary binding target