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In the context of AD, where neuroinflammation and neurotoxicity are prominent pathological hallmarks, the modus operandi of NLY01 offers a potential therapeutic avenue to mitigate analogous pathological mechanisms inherent to this neurodegenerative condition

This compound has been modified to be more effective than CBG as it can affect the peroxisome proliferator-activated receptor gamma (PPAR), which is a key molecular target for the treatment of Huntingtons disease, as well as being able to target other physiologic pathways involved in neuronal survival [163, 164]

Clin Epidemiol 12:469475

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