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Therefore, the present review explores the functional mechanisms of mitochondria in ferroptosis and discusses therapeutic strategies targeting mitochondria-mediated ferroptosis for IDD treatment, aiming to provide novel insights for clinical intervention

BPC-157 is a 15-residue, 1419.53 g/mol pentadecapeptide from human gastric juice whose literature centres on local angiogenic signalling via VEGFR2 and on nitric oxide pathway modulation

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380056) from 6 to 18 weeks of age, and co-housed with counterparts on the same diet, were purchased from The Jackson Laboratory (Bar Harbor, ME, USA)