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GHK-Cu is endogenous to the human body (found naturally in plasma, saliva, and urine), and its concentration naturally declines with age

MYCN amplification, CASP8 hypermethylation, and amplification of chromosome 17q, which encodes, amongst others, the anti-apoptotic protein Survivin, are some examples of common NB aberrations that contribute to apoptosis resistance 1,4,5

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This suggests there is biological overlap between hormone changes and incretin signaling, though the precise interactions are still being studied