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This dysfunction was associated with an impaired peroxisomal -oxidation, marked by increased levels of very long-chain fatty acids and the reduced expression of key genes involved in the -oxidation pathway such as ABCD1, ACOX1 and MFP2 (Nury et al., 2018)

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The cells treated with Sp showed slight enhancement in levels of GSH (~1.2 fold), together with minor alterations in GSSG levels, compared to untreated samples which exhibited higher GSSG content, thus indicating efficient recycling of GSSG to GSH (Fig

5g-h) and prolonged survival in mice injected with rapamycin-treated CAR T cell products from LTR (Fig