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Subsequent evaluations of blood glucose, lipid profiles, oxidative stress, histopathology, and gut microbiota were all designed to elucidate the mechanisms underlying the improvement of diabetes

In rabbit studies, GHK-Cu treatment was associated with wound contraction and granulation tissue formation

These exposures have been linked to adverse health outcomes, including potential biological and neurotoxic effects [82, 92], emphasizing the need for stringent safety protocols and exposure assessments [6]

An excess of Fe 2+ leads to the production of ROS through the Fenton reaction, which induces cell damage or death and potentially impairs organ function eventually