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Preliminary data suggest that GHK-Cu can partially reverse age-related epigenetic changes, supporting the concept of "epigenetic rejuvenation." This line of research connects GHK-Cu with the broader field of reprogramming research and cellular aging

The common link is that GLP-1s activate receptors that then work on driving down metabolic risk factors through the following mechanisms: Enhancing glucose dependent insulin secretion Slowing gastric emptying and decreasing appetite Reducing postprandial glucagon and food intake UpToDate 2 : (1) ingestion of food results in (2) release of gastrointestinal peptides (GLP-1 and GIP: glucose-dependent insulinotropic polypeptide, gastric inhibitory peptide) and (3) pancreatic beta cell hormones (insulin and amylin)