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Egr-1 functions as a master switch activated by ischemia to trigger expression of pivotal regulators of inflammation, coagulation and vascular hyperpermeability [38]

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Studies have shown that the expressions of adhesion molecules in endometrial epithelial cells increase at the receptive stage ( Our evidence confirms that a higher GLUT4 expression during the window of implantation is required for embryonic development and implantation, which is helpful in maintaining proper glucose concentration in the uterine fluid for embryo development and establishing endometrial receptivity for implantation

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