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Two downstream consequences are mechanistically central: nicotinamide is preserved for re-entry into the NAD+ salvage pathway (raising intracellular NAD+ availability), and SAM is preserved for other methylation reactions

Now, they dont look quite as robust as the data from PREDIMED study but still, any comments on that because those are relatively different

A wide-targeted urinary and serum metabolomics strategy reveals the effective substance of the Wu-tou decoction

AST/Bex also upregulated the expression of ABCA1, LRP1, and/or APOA-I, thereby facilitating cholesterol efflux partially through PPAR/RXR activation, and downregulated cholesterol biosynthesis and esterification