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The ultimate aim of any experimental system should be replication of human pathophysiology which is central to translate findings to the clinic

With the disruption of adherens junctions and tight junction proteins, the function of the intestinal epithelial barrier is further compromised, triggering bacterial translocation and a chronic inflammatory cycle, exacerbating local damage [6,7,8]

Importantly, along with an induction of HIF1, in SCD erythroblasts cultured in the absence of hemin, we detected a significantly decreased CBS expression under hypoxia compared to normoxia (Supplementary Fig