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Tumor cells often produce lipid mediators like prostaglandin E2 (PGE2) and oxidized low-density lipoprotein (oxLDL), which can suppress immune responses by impairing CD8 T cell activation and promoting M2 macrophage polarization (148). Endothelial cells within the TME are also affected by lipid accumulation and hypoxia, leading to dysfunction characterized by increased vascular endothelial growth factor (VEGF) expression and enhanced angiogenesis

Abstract Gram-negative bacteria use a plethora of virulence factors to infect eukaryotic cells

My hands and feet, which used to get cold easily during Canadian winters, seemed warmer