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As, con este entendimiento del contexto fisiopatolgico y clnico, cobra sentido preguntarse si una terapia como semaglutida con potencial de modular directamente los mecanismos neuroendocrinos del hambre y la saciedad puede realmente cambiar la historia natural de la enfermedad

Stanek MB, Borman SM, Molskness TA, Larson JM, Stouffer RL, Patton PE: Insulin and insulin-like growth factor stimulation of vascular endothelial growth factor production by luteinized granulosa cells: comparison between polycystic ovarian syndrome (PCOS) and non-PCOS women

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and holding stock in Roche Pharmaceuticals