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This was highlighted in an experimental study performed on GLP-1-treated HL-1 cardiomyocytes, in which GLP1 increased calcium transients and sarcoplasmic reticular calcium contents by regulating the expression of calcium handling proteins, leading to a reduced calcium leak and thus preventing AF development [34]

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N-terminal acetylation of antimicrobial peptide L163 improves its stability against protease degradation

This staggered approach prevents simultaneous novel stimulus on hepatic function and digestion, reducing the risk of nausea or transient liver enzyme elevation