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The reaction followed the pathway CO 2 + 2H + 2e CO + H 2 O, with holes simultaneously oxidizing H 2 O

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an environmental pollutant)-induced lipid peroxidation and cell necrosis by blocking CYP2E1-mediated CCL 4 metabolic activation and by upregulating Nrf2 downstream genes for NQO-1, HO-1, GCL, GST, and superoxide dismutase (SOD1) (67, 68)