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The GLP-1 Comparison The comparison to GLP-1 drugs is tempting but imperfect

Plaque formation begins with the accumulation of lipid-laden macrophages

Its mechanisms include: Actin polymerization mobilizes cellular building blocks for tissue repair Cell migration accelerates fibroblast and keratinocyte movement to injury sites Angiogenesis promotes new blood vessel formation via VEGF upregulation Anti-inflammatory reduces IL-1 and TNF- at injury sites TB-500's smaller molecular size gives it excellent tissue distribution throughout the body