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Blocking the cytokine-mediated inflammatory cell death signaling pathway may benefit patients with COVID-19 or other infectious diseases by limiting tissue damage ( Figure 1 Implications on Antibacterial and Antiviral Immunity According to in vitro studies, TNF- facilitates the SARS-CoV interaction with angiotensin-converting enzyme 2 (ACE2) ( Nuclear factor kappa B (NF-B), a transcription factor modulating both innate and adaptive immunity, is also crucial in the progress of COVID-19 (Figure 2)

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Pickart and Margolina (Symmetry, 2018) A 2019 in-vitro study demonstrated that GHK-Cu at 1 to 10 nM concentrations significantly increased fibroblast proliferation and type I collagen gene expression in tendon-derived cells