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Additionally, GLP-1 treatments boost the liver's production of sex hormone-binding globulin (SHBG), which binds to excess androgens, reducing their activity

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FAK-paxillin activation for fibroblast migration Growth hormone receptor upregulation on tendons VEGFR2-Akt-eNOS pathway for blood vessel formation Nitric oxide modulation reducing inflammation Erg-1 transcription factor inhibition Angiogenesis, the formation of new blood vessels, represents another critical mechanism