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The mechanism behind the potential anti-inflammatory action of GHK-Cu may be associated with its influence on the NF-B pathway, a key regulator of inflammation

Figure 8d, h shows the results of VEGF immunofluorescence staining, which revealed a greater level of VEGF in the G-RHEA@Gel group than in the control group, indicating increased blood vessel formation due to the hydrogel, oxygen delivery, and ROS scavenging

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Transient palpitations: It may be related to sympathetic nerve excitation caused by the activation of 3-AR, with an incidence rate of less than 5%