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Additional restriction to immune access is conferred by physical barriers of structural ECM remodeling, whereby tumor-derived discoidin domain receptor 1 fragments realign collagen fibers, forming physical barriers that impede immune cell penetration (177)
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We also found higher CD36 expression, but to a lesser extent than for LAR subtype, in mesenchymal-like subtype of TNBC (MSL and MES) also shown to be sensitive to ferroptosis due to their high iron uptake [22], validating the correlation between CD36 expression and ferroptosis
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