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The protection appears to result from the peptide's ability to bypass inhibitory IGFBP interactions, allowing more effective activation of IGF-1 signaling pathways that enhance sarcolemmal stability and reduce membrane damage during eccentric contractions

Am J Phys Lung Cell Mol Phys 279(6):L1005L1028 Cortassa S, ORourke B, Aon MA (2014) Redox-optimized ROS balance and the relationship between mitochondrial respiration and ROS

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