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Studies in animal models suggest semaglutide may modulate adipose tissue browning (increasing metabolic rate), regulate autophagy (the cellular process of cleaning out damaged components), and exert direct anti-inflammatory effects

Postprandial elevation of serum LPS indicates that metabolic endotoxemia induced by dietary fat may underlie the frequently observed postprandial inflammatory response

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Semaglutide is the original glucagon-like peptide-1 (GLP-1) that is supposed to be taken once weekly