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Potential Role of Chronic Physical Exercise on Mitochondrial Function and Structure: Implications on Vitiligo Development Melanocytes and PBMCs from vitiligo patients have deficient energy metabolism due to the low concentration and abnormal distribution of cardiolipin in the mETC, which prevents the stability and creation of supercomplexes (DellAnna et al., 2007)

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These reactive oxygen species may subsequently interact with superoxide dismutase and glutathione, leading to reduced levels of these antioxidants, making the brains ability to cope with oxidative stress damage much reduced and further accelerating neuronal cell damage in the corresponding brain regions ( The glutamate hypothesis of schizophrenia suggests that the function of N-methyl-D-aspartate (NMDA) receptors is impaired in this disorder