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Luthi et al

Uversky, 2002b

Our major insight from examining the vitamin B 12 GSH data in conjunction with the Reed-Nijhout model is that cysteine-block explains why GSH does not increase as a result of vitamin B 12 deficiency induced hyperhomocysteinemia

Oestrogen-dependent satellite cell activation and proliferation following a running exercise occurs via the PI 3K signalling pathway and not IGF-1