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It has been found that widespread DNA methylation abnormalities exist in the intestinal mucosal cells of IBD patients, some of which are associated with IBD susceptibility loci [196, 197]

However, while this initial hepatic fat accumulation represents a key early event, it is insufficient on its own to drive disease progression

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Despite these limitations, the majority of existing studies suggest that therapies aimed at reducing uric acid levels may potentially attenuate the progression of CKD