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Choi, M, Kadara H, Zhang J, Parra Cuentas E, Rodriguez Canales J, Gaffney SG, Zhao Z, Behrens C, Fujimoto J, Chow C, Kalhor N, Moran C, Rimm D, Swisher SG , Gibbons DL, Heymach JV, Kaftan E, Townsend J, Lynch TJ, Schlessinger J, Lee JJ, Lifton R, Herbst RS, Wistuba II

Esophagectomy after concurrent chemoradiotherapy improves locoregional control in clinical stage II or III esophageal cancer patients

The cycle does not feel like a transformation

Chronic hyperglycemia-induced ROS overproduction, primarily derived from mitochondrial ETC leakage and NOX activation, leads to eNOS uncoupling due to BH4 oxidation, reducing NO bioavailability and impairing vasodilation ( 2 and mitochondrial ROS-mediated apoptosis ( Furthermore, due to a decrease in their destruction rate and/or a reduction in the production of CAT, SOD, and GPx, excess ROS promotes the deleterious changes associated with OS