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Influenza Viruses In mice following intranasal infection with a lethal dose of influenza A virus A/PR/8, the use of N-acetylcysteine increased survival rate from 58% (ribavirin monotherapy) to 92% (N-acetylcysteine and ribavirin), suggesting the antioxidants role in improving host defense mechanism and preventing pathogenesis of lung inflammation

Precursors like N-acetylcysteine (NAC) can support production but don't restore depleted levels rapidly

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It decreases COX-2 gene expression, reduces myeloperoxidase activity, and lowers pro-inflammatory cytokines including IL-6 and TNF-alpha