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As an example, during the evaluation of a patient whose primary phenotype was attention-deficit/hyperactivity disorder (ADHD), an incidental finding revealed a pathogenic nonsense mutation in SCN5A , associated with Brugada syndrome

ALC performs a variety of crucial functions, such as promoting acetyl CoA absorption into mitochondria during oxidation of fatty acid, increasing acetylcholine synthesis, accelerating protein and transmembrane phospholipid formation, and preventing undue cellular injury in the neuron (Di Cesare Mannelli et al., 2011)

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