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Increased Angiogenesis at the Injury Site: New capillary network formation accelerates in BPC-157-treated connective tissue through VEGF upregulation, supplying the vascularization required for sustained cellular repair activity

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In summary, PLB significantly improved the survival, mitochondrial function and antioxidant capacity of KGN cells, as well as suppressed cell damage and ferroptosis by inhibiting YTHDF1 expression