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J Lipid Res 2012;53:44655

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This process liberates NF-B dimers, enabling their nuclear translocation and subsequent binding to target gene promoters, thereby activating transcription of a broad array of inflammation-related genesincluding pro-inflammatory cytokines (e.g., TNF-, IL-1), anti-apoptotic molecules (e.g., Bcl-xL), and inflammatory enzymes (e.g., cyclooxygenase-2, COX-2)ultimately contributing to immune defense, cell survival, and amplification of inflammatory responses [211, 212]

Wei CH, Liu X, Gao Y, Wu YP, Guo XX, Ying Y, Wen Y, Yang HF