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In summary, this study revealed that dysfunction of cellular transport of cytoplasmic FA to mitochondria plays a critical role in UA-induced cardiomyocyte injury and thereby promotes LVR development and identified CPT1B as the key factor influenced by UA

Fasting insulin or insulin-resistance risk when appropriate

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This anatomical shift can influence how the gut responds to GLP-1 medications, which work partly by slowing gastric emptying and modulating bile acid signaling pathways